Neurodegenerative diseases are strongly associated with the buildup of protein aggregates, misfolded or altered amyloid-β, tau, α-synuclein, and so forth, wherein one altered molecule can encourage others to also alter, leading to solid deposits in brain tissue and a surrounding halo of toxic biochemistry that degrades cell function and kills cells. Much of the Alzheimer’s research and development to date has focused on how to clear those aggregates; unfortunately success in clearance of amyloid-β in Alzheimer’s disease has <a href="https://www.fightaging.org/archives/…
Supporting Evidence for Senolytics to be an Effective Treatment for Alzheimer's Disease
Neurodegenerative diseases are strongly associated with the buildup of protein aggregates, misfolded or altered amyloid-β, tau, α-synuclein, and so forth, wherein one altered molecule can encourage others to also alter, leading to solid deposits in brain tissue and a surrounding halo of toxic biochemistry that degrades cell function and kills cells. Much of the Alzheimer’s research and development to date has focused on how to clear those aggregates; unfortunately success in clearance of amyloid-β in Alzheimer’s disease has <a href="https://www.fightaging.org/archives/…