Source https://www.fightaging.org/archives/2020/08/age-related-downregulation-of-rubicon-causes-excessive-autophagy-in-adipocytes-contributing-to-metabolic-dysfunction/
Autophagy is a vital collection of cellular maintenance processes in which proteins and structures are broken down and recycled for their component parts. In short-lived laboratory species, dysfunctional autophagy shortens life span, while increased operation and efficiency of autophagy – as occurs in response to forms of stress such as heat, exercise, and calorie restriction – slows aging and extends life span.
The usual high level view of aging and autophagy is that autophagic activity declines with age, and that this loss of function contributes to cell and tissue dysfunction, and thus also to age-related disease and mortality. The <a href="https://www.fightaging.org/archives/2020/07/autophagy-in-cardiovascular-aging-is-a-complicated…
Source https://www.fightaging.org/archives/2020/08/age-related-downregulation-of-rubicon-causes-excessive-autophagy-in-adipocytes-contributing-to-metabolic-dysfunction/
Autophagy is a vital collection of cellular maintenance processes in which proteins and structures are broken down and recycled for their component parts. In short-lived laboratory species, dysfunctional autophagy shortens life span, while increased operation and efficiency of autophagy – as occurs in response to forms of stress such as heat, exercise, and calorie restriction – slows aging and extends life span.
The usual high level view of aging and autophagy is that autophagic activity declines with age, and that this loss of function contributes to cell and tissue dysfunction, and thus also to age-related disease and mortality. The <a href="https://www.fightaging.org/archives/2020/07/autophagy-in-cardiovascular-aging-is-a-complicated…
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