Source https://www.fightaging.org/archives/2021/09/successfully-treating-fibrosis-in-mice-via-the-senolytic-strategy-of-bcl-2-inhibition/
It has to be said, today’s research materials make for a fascinating read. A group of scientists, in 2021, a decade into the general acceptance of the importance of cellular senescence as a phenomenon, conducts a study of lung fibrosis in which they achieve a reversal of that fibrosis using a bcl-2 inhibitor, venetoclax, and then publish a paper that fails to mention cellular senescence even once.
Fibrosis is a dysfunction of tissue maintenance, producing scar-like collagen deposits that disrupt tissue function. There is a weight of evidence for fibrosis as a phenomenon to be <a href="https://www.fightaging.org/archives/2020/01/a-start-on-establishing-how-senescent-cells-drive-fibrosis-in…
Source https://www.fightaging.org/archives/2021/09/successfully-treating-fibrosis-in-mice-via-the-senolytic-strategy-of-bcl-2-inhibition/
It has to be said, today’s research materials make for a fascinating read. A group of scientists, in 2021, a decade into the general acceptance of the importance of cellular senescence as a phenomenon, conducts a study of lung fibrosis in which they achieve a reversal of that fibrosis using a bcl-2 inhibitor, venetoclax, and then publish a paper that fails to mention cellular senescence even once.
Fibrosis is a dysfunction of tissue maintenance, producing scar-like collagen deposits that disrupt tissue function. There is a weight of evidence for fibrosis as a phenomenon to be <a href="https://www.fightaging.org/archives/2020/01/a-start-on-establishing-how-senescent-cells-drive-fibrosis-in…
What Do You Think?
comments